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Aluminum's Health Risks Summarized in New Systematic Review

So I recently launched my inaugural book, LIFE: The Epic Story of Our Mitochondria, during a presentation to about 150 colleagues at the CHFA East show in Toronto. The event was a big success and I want to thank everyone for their support, glowing reviews, and excitement over the future of mitochondrial medicine.

As you may have guessed from above, the topic of my book is mitochondrial health and cellular energetics. It's an incredibly fascinating area with a tonne of research being published every week. Seriously. I'm talking on average about 300 new peer-reviewed publications (every week) that are in one way or another connected to mitochondria. That's insane! Anyway, I think you'll hear MUCH more about the topic in the near future, so if you want to get your hands on a copy of the book and give yourself a head start in understanding this rapidly growing area, you can buy the book online through these various retailers:
  • Zwell.ca - my exclusive online healthfood retailer for the softcover copy
  • Amazon - where you can also purchase the Kindle ebook version
  • Chapters/Indigo - where you can buy the Kobo ebook version
  • many health food stores across Canada will soon stock the book in their literature section

I mention this since today's new study of discussion partly looked at the mitochondria, in relation to aluminum exposure. So, with that said, let's talk about this new study...


"Low" Doses May Be Effective for Vitamin K2 (as MK-4)

It's been a while since a new vitamin K study has been published. Now here is a study that suggests we can still get the bone benefits of MK-4 (a form of vitamin K2) at much lower doses than previously studied (which was about 45,000 mcg/day, and up to 60,000 mcg/day). For this new study, researchers recruited 15 healthy men (average age of 25) to participate in this newly published non-placebo controlled dose-finding study.


CoQ10 and Low Birth Weight's Future Complications

Those born with low birth weight will typically experience rapid postnatal growth that allows these individuals to catch up to their peers. Although the underlying mechanisms are poorly understood, studies indicate that low birth weight followed by rapid growth increases the risk of cardiovascular-disease (CVD) as adults.


Mitochondria Behind Age-Related Exercise-Induced Fatigability

With age, we tend to see an increase in "fatigability" while the capacity for mitochondrial energy production decreases. Logically, this correlation makes sense; diminished mitochondrial function may contribute to higher levels of fatigability the older we get.

To confirm whether this is truly due to mitochondrial function (or dysfunction as we age), researchers investigated the relationship between fatigability and skeletal muscle mitochondrial function in 30 participants with an average age of 78.5 years. Fatigability was defined using rating of perceived exertion after a 5-minute treadmill walk at 0.72 m/s. Phosphocreatine recovery in the quadriceps was measured using magnetic resonance spectroscopy and images of the quadriceps were captured to calculate quadriceps volume. ATPmax (amount of ATP--the energy currency in cells--replenished per second) and oxidative capacity (the capacity to product energy via aerobic respiration in the mitochondria) of the quadriceps (ATPmax·Quadriceps volume) were calculated. Peak aerobic capacity (VO2peak) was measured using a modified Balke protocol.