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N-Acetyl-Glucosamine May Stop Multiple Sclerosis

Before I get to today's study, take a look at this article regarding the new crime bill our Canadian Conservative government, under Stephen Harper (AKA. a smarter version of George W. Bush, for those unfamiliar with Canadian politics) is about to push through into law.
Potheads Fare Worse Than Child Rapists in Canadian Crime Bill

On the way to the office earlier this week, one of the radio stations I listen to while driving had Theo Fleury as their guest, who said current stats show 1 in 3 girls and 1 in 6 boys will be sexually molested by the time they're 18 years old.
...and Stephen Harper is more worried about a relatively benign botanical that actually has incredible medicinal properties, than protecting children. Fail.

Ok, now...

Current treatments and emerging oral therapies for Multiple Sclerosis (MS) are limited by ineffectiveness, cost and/or toxicity. Genetic and environmental factors alter the branching of certain sugars and result in T cell hyperactivity,  which promotes spontaneous inflammatory demyelination and neurodegeneration seen in MS. The compound N-acetylglucosamine (NAG) enhances branching and inhibits T cell activity.

In this newly published animal study, the researchers report that oral NAG inhibits multiple mechanisms that lead to MS-symptoms, and as a result attenuates the clinical severity of MS-like symptoms and it's progression. The data suggests that oral NAG may provide an inexpensive and non-toxic oral therapeutic agent for MS that directly targets an underlying molecular mechanism causal of disease.

Source: N-acetylglucosamine inhibits T-helper 1 (Th1) / T-helper 17 (Th17) responses and treats experimental autoimmune encephalomyelitis

For my Canadian readers, have an amazing Thanksgiving long weekend, and truly take a moment to think about all those things we should be thankful for. For real.

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